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ECGcheatsheet5 PDF

This document provides guidance on interpreting an electrocardiogram (ECG). It outlines normal and abnormal findings including: - Common rates and rhythms seen on ECG as well as how to analyze axis, hypertrophy, and waves/intervals. - Characteristics of a sharp or diffuse J point as well as signs of atrial and ventricular enlargement. - Patterns associated with different types of myocardial infarction and how STEMI evolves over time. - Abnormal findings indicative of conditions like ischemia, hyperkalemia, left and right bundle branch blocks, and more.

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Miko Ramoso
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301 views1 page

ECGcheatsheet5 PDF

This document provides guidance on interpreting an electrocardiogram (ECG). It outlines normal and abnormal findings including: - Common rates and rhythms seen on ECG as well as how to analyze axis, hypertrophy, and waves/intervals. - Characteristics of a sharp or diffuse J point as well as signs of atrial and ventricular enlargement. - Patterns associated with different types of myocardial infarction and how STEMI evolves over time. - Abnormal findings indicative of conditions like ischemia, hyperkalemia, left and right bundle branch blocks, and more.

Uploaded by

Miko Ramoso
Copyright
© © All Rights Reserved
We take content rights seriously. If you suspect this is your content, claim it here.
Available Formats
Download as PDF, TXT or read online on Scribd
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RATE

300 150 100

75

60

50

SHARP J POINT
ST seg. & T wave well demarcated, not merged as in STE
J point elevation is normal in young, healthy athletes
DIFFUSE J POINT
ST slowly curving with only an area J point can be found

43

- Count number of complexes x 6 (standard ECG = 10sec)


RHYTHM
Locate the P wave (rate, axis, morphology)
What is the relationship between the P wave and QRS?
Analyze QRS morphology
AXIS DEVIATION
Normal (-30 to 90)
Left
Right

Lead I QRS
+
+
-

Lead II/aVF QRS


+
+

HYPERTROPHY
LEFT ATRIAL ENLARGEMENT (P mitrale)
P wave > 0.12sec and bifid in lead II
RIGHT ATRIAL ENLARGEMENT (P pulmonale)
P wave > 0.25mV in lead II
LVH
R wave in V5 or V6 >25mm
S wave in V1 or V2 >25mm
Sum of R wave in V5 or V6 + S wave in V1 >35mm
RVH
R wave > S wave in V1

LAE

RAE

5mm=0.2sec(200ms)
ST segment

0.12s<PR<0.2s

Q S
QRS<0.12s
QT<(1/2)RR
QTc=QT/sqr(RR)

1mm
=0.1mV

LVH

RVH

QTc interval
W
<0.45
>0.47

NORMAL Q WAVES
Small (septal) q waves normal in leads aVL, I, II, V5, V6
Can be normal on expiration in lead III
PATHOLOGICAL Q WAVES (PRIOR MI)
>1-2 small squares deep (or >25% of R wave)
>1 small square wide (or 30ms)
More likely diagnostic if with inverted T wave
DOMINANT R WAVE
In lead V1: normal in young children; seen in RVH, RBB, HCM, posterior MI
In lead aVR: TCA poisoning, dextrocardia, VT
POOR R WAVE PROGRESSION
Prior anteroseptal MI, cardiomyopathy, LVH, RVH/COPD, LBBB

PATTERNS
Anterior MI (LAD) = V1-V4
Lateral MI (LCx) = I, aVL, V5-V6
Anterolateral MI (LAD) = I, aVL, V1-V6
Inferior MI (RCA, LCx) = II, III, aVF
Inferolateral MI (RCA, LCx) = I, aVL, V5-V6, II, III, aVF
Acute posterior MI (RCA or LCx):
Dominant R waves in leads V1-V3
ST depression in V1-V3
Upright, tall T waves
STEMI EVOLUTION
Hyperacute T waves (tall, peaked,
symmetric)
STE in contiguous leads (concave
convex, merging with T wave)
Development of Q wave and T wave
inversions as ST returns to baseline

1mm=
0.04sec(40ms)

1-15yo
M
Normal
0.44 <0.43
Prolonged >0.46 >0.45
(upper 1%)

NORMAL ECG

(Known LBBB and pacing make ECG less diagnostic for ACS)

WAVES, INTERVALS, & SEGMENTS

ANTERIOR ST DEP.
ST SEGMENT ELEVATION
POSTERIOR MI
WITH ANGINA
(New STE at the J point)
In all leads (except V2-V3), significant STE =
In two contiguous leads
0.1mV
In leads V2-V3, significant STE =
0.15mV in women
0.2mV in men 40yo
0.25mV in men 40yo
ST SEGMENT DEPRESSION
(New horizontal or down-sloping STD)
Significant STD =
In two contiguous leads
0.05mV
and/or
T-wave inversion 0.1mV in two contiguous leads with
Prominent R wave or R/S ratio>1

HYPERKALEMIA
NORMAL INVERTED T WAVES
Normal in leads aVR, V1
Can be normal in lead V2 in young pts,
lead V3 in black pts, lead III during
expiration
INVERTED T WAVES IN ISCHEMIA
0.1mV in two contiguous leads
TALL T WAVES
<1/2 preceding QRS
LVH LV STRAIN PATTERN TWI in leads I, aVL, V5-6
RVH RV STRAIN PATTERN TWI in leads II, III, aVF

BBB

ANTERIOR MI WITH TALL T WAVES

V1: M
V6: W

V1: W
V6: M

SOURCES: ECG tutorials on UpToDate (Basic principles of ECG analysis, Myocardial ischemia and infarction),
Making Sense of the ECG by Houghton, Pocket Medicine by Sabatine; Third Universal Definition of Myocardial
Infarction by Thygesen et al; lifeinthefastlane.com; compiled by Henry Del Rosario

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